Har gått till en reumatolog och skall senare få tid till en sjuksköterska som har docerat i immunologi. Under tiden så har jag luskat lite själv om cytokiner, eftersom Imurel inte heller fungerade att bryta igenom mitt immunförsvar.
Idag har jag fått kontakt med Örtspecialist som kan ta in Hydrangea( (Dichroa Febrifuga) på kinesiska(Chang Shan) utifrån en del av de rapporter som jag fick nys om här på forumet:
The Promise of Halofluginone
Mammals generate several types of CD4+ T helper cells, including TH1, TH2, Treg, and TH17 cells. TH1 and TH2 cells secrete cytokines that activate immune responses to bacteria and other infectious agents (TH1) or as part of an allergic response (TH2); Treg cells prevent other T cells from attacking the body. TH17 cells mediate immune responses to certain bacteria and fungi by producing the proinflammatory cytokine interleukin-17 (IL17) (Blander and Amsen 2009). However, TH17 cells are gaining recognition as important and broad mediators of autoimmunity. Hence autoimmune disease researchers are keenly interested in discovering drugs that specifically target TH17 cells. Halofuginone, an herb used in traditional Chinese medicine and to treat malaria and scleroderma, comes close to being such a drug. According to Sundrud and his team, halofuginone selectively inhibits the differentiation of mouse and human TH17 cells but has only minor effects on TH1, TH2, and Treg cell lineages. Unfortunately, halofuginone does not affect the function of already developed TH17 cells, a potential drawback to its use as an MS therapy because autoimmune disorders usually manifest themselves after autoaggressive T cells develop. Furthermore, TH17 cells produce IL10 and IL22, both of which function in immune tolerance and tissue repair (Blander and Amsen 2009). Nevertheless, the benefits of halofuginone may well outweigh its shortcomings, especially if its utility can be expanded to prevent MS, a disease that affects so many people and for which few therapies currently exist.